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Thetranslational pathways involved in this Treg defect will be discussed in detail later in thischapter, but suffice it to say, this is just one aspect of a complex series of events that lead tothe development of a myriad of autoimmune diseases associated with Treg dysfunction.These dysfunctions of Tregs can not only originate from the Treg itself, but can also bemanifested in T cell resistance (mentioned above and in a future chapter concerning Tregeffector mechanisms), cytokine insensitivity, or just inadequate numbers of Tregs (Buckner2010) as in the case with the global expansion of anti-melanocyte CD8 effector T cells invitiligo.

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TV and video games should only be apart of the child’s play and parents should monitor contentand amount of time spent in use. These FOXO3 proteins have the ability to activate the genetic expression all ofCDKIs mentioned above (Hendrick 2012). The mechanismof this complication is not understood; nospecific antidote is available.

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