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Thetranslational pathways involved in this Treg defect will be discussed in detail later in thischapter, but suffice it to say, this is just one aspect of a complex series of events that lead tothe development of a myriad of autoimmune diseases associated with Treg dysfunction.These dysfunctions of Tregs can not only originate from the Treg itself, but can also bemanifested in T cell resistance (mentioned above and in a future chapter concerning Tregeffector mechanisms), cytokine insensitivity, or just inadequate numbers of Tregs (Buckner2010) as in the case with the global expansion of anti-melanocyte CD8 effector T cells invitiligo.

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